Gastrointestinal Problems

 Dysbiosis

Dysbiosis—is the state of disordered microbial ecology that causes disease. It may exist in the mouth, gastrointestinal tract or vaginal cavity. A common cause of bacterial or fungal dysbiosis is repeated or long term use of antibiotics. Antibiotics kill both the bacteria you want to kill and the friendly bacteria in the intestine and vagina. This leaves these areas open to be colonized by yeast, unfriendly bacteria and parasites. In dysbiosis, unfriendly organisms induce disease by altering the nutrition, the immune response and causing inflammation and irritation in the intestines and vagina.

Putrefaction– results from diets high in fat and meat and low in fiber. Altered body structure causes a failure to properly chew the food and insufficient digestive chemistry.  This leaves chunks of meat in the digestive tract which only the surface area can be affected by the diminished digestive chemistry. The chunks then provide a food source for bacteria, which produce ammonia increasing fecal pH. Increased pH raises your risk for colon and breast cancer. Enzyme activity changes from constructive to destructive causing arthritis and degenerative joint disease. Estrogen scheduled for elimination is re-activated raising blood estrogen levels.

Fermentation Excess— excess carbohydrate consumption and intolerance to sugars induce excess bacterial fermentation usually resulting from small intestine bacterial overgrowth. The by-product of fermentation is gas. Abdominal bloating or distention, flatulence, diarrhea, constipation and feelings of malaise are commonly described. In small intestine bacterial overgrowth, degradation of intestinal brush-border and pancreatic enzymes by bacterial protease may cause maldigestion.

Fecal short chain fatty acids may be elevated. People with fermentations excess are usually intolerant of soluble fiber supplements  and often benefit from antimicrobials and reduction of carbohydrate consumption.

Deficiency - Exposure to antibiotics or a diet depleted of soluble fiber may create a deficiency of normal fecal flora, including Bifidobacteria, Lactobacillus and E. coli. Direct evidence of this condition is seen in stool cultures when concentrations of any of these organisms are reduced. This condition has been described in patients with irritable bowel syndrome and food intolerance.

Deficiency and Putrefaction Dysbiosis often occur together and respond to the same treatment. Probiotic supplementation as well as fructooligosaccharides is often helpful in reestablishing a normal flora.

Sensitization - Abnormal immune responses to components of the normal indigenous intestinal microflora may contribute to the onset of inflammatory bowel disease, arthritic conditions, connective tissue disorders, scleroderma and other skin conditions such as psoriasis or acne. Endotoxins may activate the alternative complement pathway, and sensitization may complement fermentation excess. Similar treatment may benefit both conditions

Pain, Inflammation, and Oxidative Stress - An adverse antigen (food that you predominantly eat remaining in the gut undigested due to a lack of stomach chemistry) that interacts with the Peyers Patchs [gut-associated lymphoid tissue (GALT)] will trigger an inflammatory response that will create a vicious cycle which includes inflammation, pain and oxidative stress

 Resulting in sensitization to any food you predominantly eat

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